Indigo V.L. Rose

UCSF Neuroscience PhD Candidate

Complement 3+-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia


Journal article


K. Hartmann, D. Sepulveda-Falla, ***IVL. Rose, C. Madore, C. Muth, J. Matschke, O. Butovsky, SA. Liddelow, M. Glatzel, S. Krasemann
Acta Neuropathologica Communications, 2019

Semantic Scholar DOI PubMedCentral PubMed
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APA   Click to copy
Hartmann, K., Sepulveda-Falla, D., Rose, ***I. V. L., Madore, C., Muth, C., Matschke, J., … Krasemann, S. (2019). Complement 3+-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia. Acta Neuropathologica Communications.


Chicago/Turabian   Click to copy
Hartmann, K., D. Sepulveda-Falla, ***IVL. Rose, C. Madore, C. Muth, J. Matschke, O. Butovsky, SA. Liddelow, M. Glatzel, and S. Krasemann. “Complement 3+-Astrocytes Are Highly Abundant in Prion Diseases, but Their Abolishment Led to an Accelerated Disease Course and Early Dysregulation of Microglia.” Acta Neuropathologica Communications (2019).


MLA   Click to copy
Hartmann, K., et al. “Complement 3+-Astrocytes Are Highly Abundant in Prion Diseases, but Their Abolishment Led to an Accelerated Disease Course and Early Dysregulation of Microglia.” Acta Neuropathologica Communications, 2019.


BibTeX   Click to copy

@article{k2019a,
  title = {Complement 3+-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia},
  year = {2019},
  journal = {Acta Neuropathologica Communications},
  author = {Hartmann, K. and Sepulveda-Falla, D. and Rose, ***IVL. and Madore, C. and Muth, C. and Matschke, J. and Butovsky, O. and Liddelow, SA. and Glatzel, M. and Krasemann, S.}
}